Parasitic Diseases List

Shellye's story Women have hemophilia, too. Hemophilia A (factor VIII [8] deficiency) is in five generations of my family—we traced it from my great grandfather down to my nephew. Hemophilia is not scary to us, as it is something we grew up around. My dad had severe hemophilia A, and I remember helping him infuse (inject medicine into a vein) even as a young child. I would hand him his supplies and was curious and interested in what he did to take care of himself. Growing up, we understood that men had hemophilia and women were "carriers." Women passed along the X-linked gene but did not actually get hemophilia because (it was believed) that the "good X chromosome" compensated for the X chromosome that carried hemophilia. When women in our family struggled with bleeding issues, hemophilia was not even on the radar as a possible contributor or cause. When I was in school, I hated physical education classes, and I mean HATED them. When I ran, it hurt a lot

Abstract Detection of high glycated hemoglobin (A1c) is associated with worse postoperative outcomes, including predisposition to develop systemic and local infectious events. Diabetes and infectious Outcomes in Cardiac Surgery (DOCS) study is a retrospective case–control study aimed to assess in DM and non-DM cardiac surgery patients if a new screening and management model, consisting of systematic A1c evaluation followed by a specialized DM consult, could reduce perioperative infections and 30-days mortality. Effective July 2021, all patients admitted to the cardiac surgery of IRCCS ISMETT were tested for A1c. According to the new protocol, glucose values of patients with A1c ≥ 6% or with known diabetes were monitored. The diabetes team was activated to manage therapy daily until discharge or provide indications for the diagnostic-therapeutic process. Propensity score was used to match 573 patients managed according to the new protocol (the Screen+ Group) to 573 patients admitted bef

Author Kwame Donkor, MD Staff Physician, Department of Emergency Medicine, Olive View Internal Medicine, University of California Los Angeles Medical Center Kwame Donkor, MD is a member of the following medical societies: American College of Emergency Physicians Disclosure: Nothing to disclose. Coauthor(s) Scott Lundberg, MD Assistant Clinical Professor of Medicine, UCLA School of Medicine; Consulting Staff, Departments of Medicine and Emergency Medicine, Olive View Medical Center Scott Lundberg, MD is a member of the following medical societies: American College of Emergency Physicians, American College of Physicians, Society of General Internal Medicine, Association of Program Directors in Internal Medicine Disclosure: Nothing to disclose. Specialty Editor Board Francisco Talavera, PharmD, PhD Adjunct Assistant Professor, University of Nebraska Med

Introduction The role of microbiota in health and disease among humans has recently been demonstrated using molecular methods (Cho and Blaser, 2012; Siqueira and Rocas, 2017; Tsuji et al., 2018). The oral microbiota was no exception, and its disruption has been linked to a various range of oral diseases including gingivitis and periodontitis (Lourenco et al., 2014; Lamont et al., 2018; Santi-Rocca, 2020). The complex interactions of different resident microbes that result in an equilibrium to maintain the healthy state of the oral cavity is termed oral eubiosis (Lamont et al., 2018). In contrast to eubiosis, the disruption in oral microbiota's homeostatic state is referred to as oral dysbiosis (Darveau, 2010; Lourenco et al., 2014; Kinane et al., 2017). Periodontal disease represents a state of chronic inflammation in gingiva, bone and supporting ligaments, with gingivitis and periodontitis as the most common presentations (Di B